Of note, miR-137 expression was lowest in SW480 and SW620 tumor cells, which conversely exhibited the highest ASCT2 abundance, whereas NCM460 cells with the highest miR-137 expression showed a much lower ASCT2 protein level (Figure 3a), suggesting miR-137 as an essential regulator of endogenous ASCT2 expression. The gene discussed is SLC1A5; the disease is neoplasm.