Furthermore, IFN-γ production by Th2/1 cells generated during helminth infection often including tissue migratory larval stages may be advantageous in preventing bacterial dissemination facilitated by tissue damage (Pesce et al., 2008), especially upon parasite re-exposure of organs densely seeded with Th2 memory cells (Steinfelder et al., 2017). Here, IFNG is linked to helminthiasis.