In patients with DN, microalbuminuria and proteinuria are known to be mainly subsequent to glomerular injury, which is generated by abnormalities in the glomerular endothelium and increased permeability of the capillary wall, thereby causing the loss of high‐molecular‐weight proteins (proteins with molecular weight >65 kDa) and albumin in urine. Here, ALB is linked to liver dysplastic nodule.