As shown in Figure 8A, API treatment was able to reduce the expression of EGFR in human MM cells (MM-F1, p = 0.0091; MM-B1, p = 0.0035; H-Meso-1, p = 0.0014) and of ErbB2 in human and mouse MM cell lines (MM-F1, p = 0.013; MM-B1, p = 0.0005; H-Meso-1, p = 0.007; #40a, p = 0.0024). Here, EGFR is linked to Miyoshi myopathy.