In an attempt to investigate if the 26S proteasomal pathway affects the turnover of ANKRD1, Badi et al., treated HeLa cells (stably transfected with ANKRD1) or SK-MES-1 cells (human lung carcinoma) with the protein synthesis inhibitor cycloheximide (CHX) in the absence or presence of MG132, a 26S proteasome inhibitor [106]. This evidence concerns the gene ANKRD1 and lung carcinoma.