Bax has been suggested to be required for Drp1-mediated mitochondrial fission caused by photodynamic therapy in human lung adenocarcinoma cells [76], while Drp1 influenced Bax translocation to mitochondria in response to irradiation-induced apoptosis [77] and Drp1-induced membrane remodeling stimulates Bax oligomerization [78]. Here, BAX is linked to lung adenocarcinoma.