CaMK2 modulations (mainly “inhibition”) are therefore being exhaustively researched for anti-tumor therapeutics in breast, prostate, osteosarcoma, liver and CML cancers though its targeting in brain cancers is not yet robustly studied (Li and Hanahan, 2013; Pellicena and Schulman, 2014; Wang et al., 2015; Chi et al., 2016). This evidence concerns the gene CAMK2B and neoplasm.