Idiopathic recurrent pericarditis is currently considered an autoinflammatory disease based on its distinctive features such as its relapsing course with episodes of fever and high inflammatory markers [C-reactive protein (CRP), erythrocyte sedimentation rate (ESR)] in the absence of specific autoantibodies or autoreactive T cells, its serosal involvement (pericardial and occasionally pleural), and its response to colchicine and, more importantly, to IL-1 inhibition (6, 10, 11). Here, CRP is linked to pericarditis.