In addition to the RET, the superexpression of receptors 2 and 3 of the vascular endothelial growth factor (VEGF), which leads to tumor angiogenesis and nutrition, and of the epidermal growth factor receptor (EGFR), involved in tumor proliferation, also contributes to the pathogenesis of MTC (3). This evidence concerns the gene VEGFA and medullary thyroid gland carcinoma.