Four weeks after MI, the echocardiography and hemodynamics were measured as the data of cardiac function of MI mice, which were then anesthetized for the collection of blood for serum NO, eNOS, Ang II, SOD and MDA measurements and the harvest of hearts for histological analysis, as well as the measurement of AngII, pro-inflammatory cytokines and anti-inflammatory cytokines in cardiac tissues. This evidence concerns the gene SOD1 and myocardial infarction.