Tokmak et al. [14], reported that AMH level (at delivery) was lower in preeclamptic patients than in normal pregnant women and there was no relationship between AMH level and adverse maternal (eclampsia, persistent hypertension and hemolysis, elevated liver enzyme and low platelets, and perinatal outcomes (prematurity, hypoglycemia, polycythemia, respiratory distress syndrome and perinatal deaths). This evidence concerns the gene AMH and hemolysis.