Experiments involving a DTA-1 devoid of FcRγ binding capacities have shown that it is ineffective in controlling tumor growth (Bulliard et al. 2013), strongly suggesting that the antibody-mediated cytotoxicity of DTA-1 plays a predominant role in shifting an initial intra-tumor balance between the Tregs and Teffs in favor of the latter. Here, FCER1G is linked to neoplasm.