NR3C2 and persistent truncus arteriosus: Although EC‐MR−/− and EC‐MR+/+ mice were exposed to the same degree of LV pressure overload and the development of similar cardiac hypertrophy and fibrosis, EC‐MR−/− mice displayed an ejection fraction and fractional shortening very similar to Sham operated mice and preserved as compared to EC‐MR+/+ mice exposed to TAC.