The other gene expression analyses on DFs reported in the literature are not comparable with our study because, regardless of tumor CTNNB1 mutational status, they compared DFs with nodular fasciitis, normal tissues, and/or solitary fibrous tumors in order to find specific gene signatures associated with DF biology or outcome (Bacac et al., 2006; Colombo et al., 2011; Denys et al., 2004; Salas et al., 2010; Skubitz and Skubitz, 2004). Here, CTNNB1 is linked to solitary fibrous tumor.