The increased survival of miR-128-3p-overexpressing NSCLC cells under drug treatment was closely associated with miR-128-3p-induced anti-apoptotic effect, as demonstrated by miR-128-3p-induced decrease of TUNEL-positive cells and suppression of Caspase 3 and PARP activation (Fig. 2h,i), as well as abrogation of cellular uptake of the chemotherapeutic drugs(Supplementary Fig. 3h). Here, CASP3 is linked to non-small cell lung carcinoma.