Alzheimer’s disease (AD), similar to other neurodegenerative diseases, is characterized by the accumulation of protein aggregates, namely amyloid β-peptide (Aβ) and Tau tangles, which lead to synaptic and neuronal damage, particularly in the hippocampal and the inferior parietal lobule (IPL) regions of the brain, resulting in memory loss (Braak and Braak, 1991; Borlikova et al., 2013). This evidence concerns the gene MAPT and Alzheimer disease.