The hypothesis that EMT might also contribute to immune escape of tumors was addressed by exploiting the human mammary carcinoma model MCF7 which underwent EMT, following stable expression of SNAIL or after prolonged exposure to tumor necrosis factor alpha (TNF‐α), exhibited reduced susceptibility to CTL‐mediated lysis (Akalay et al., 2013). This evidence concerns the gene TNF and breast carcinoma.