AXL and melanoma: A recent elegant single‐cell analysis revealed that all melanomas contain AXL‐high populations to a certain degree and it is now well accepted that the AXL‐high phenotype is linked to MAPKi therapy resistance and that there is an enrichment of the AXL‐high phenotype in BRAFi/MEKi therapy post‐relapse melanomas (O'Connell et al, 2013; Anastas et al, 2014; Muller et al, 2014; Ji et al, 2015; Tirosh et al, 2016).