In the presence of active TGF‐β1, during a murine study of bleomycin‐induced lung fibrosis, epithelial integrin and focal adhesion kinases up‐regulated Src activity and Src inhibition attenuated EMT, which is a potential source of myofibroblasts accumulation in fibrotic lungs 42, 43, 44. Here, TGFB1 is linked to pulmonary fibrosis.