A recent study reported on the important roles of the reprogramming of cellular metabolism in tumor metastasis.9 Based on the mitochondrial localization of FAM210B and loss of the FAM210B effect on cancer metastasis, we measured its oxygen consumption rates (OCRs) and extracellular acidification rates (ECARs) in the presence or absence of distinct inhibitors. The gene discussed is MIMS2; the disease is neoplasm.