MiR-98 overexpression was shown to suppress tumor cell proliferation [11,12], epithelial-mesenchymal transition (EMT), chemoresistance [13], and metastasis [14] by targeting p21 (RAC1)-activated kinase 1, SNAIL, and LIN28 [15,16,17], while its inhibition leads to tumor metastasis and poor clinical outcome [14,16]. Here, PAK1 is linked to neoplasm.