KRAS and neoplasm: Noteworthy, this correlation between low-expressing autophagy, KRAS-activating mutations and poor clinical outcome was still observed when tumours were ranked for the mean expression of the entire subset of autophagy genes identified in the primary fly screen (Supplementary Figure 4), although this effect was mostly, but not exclusively, driven by the GABARAP family and VAMP2 (Supplementary Table 3).