PRKN and pulmonary fibrosis: This enhanced accumulation of inflammatory cells in BLM-treated PARK2 KO mice was reduced by treatment with PFD, which was comparable to levels seen in BLM-treated wild type mice with PFD treatment (Fig. 5e), suggesting that not only inhibition of myofibroblast differentiation but also regulation of inflammation can play a role in PFD-mediated attenuation of BLM-induced lung fibrosis.