No mutations in Lats2 were observed in this study, however 20% of samples in this study contained a copy number deletion of Lats2. With post-transcriptional modification potentially accounting for loss of these tumour suppressors in the mouse asbestos induced MM model it is not possible to conclusively say that Cdkn2a deletion is an independent driver of MM. The gene discussed is CDKN2A; the disease is neoplasm.