EGFR and glioblastoma: In contrast to EGFR- or EGFRvIII-targeted monospecific NK-92 variants, dual targeting of EGFR and EGFRvIII with a cetuximab-based CAR recognizing a common epitope of the receptors, thereby circumvented immune escape in mixed tumors that similar to the clinical situation, consisted of EGFR-positive and EGFR/EGFRvIII-double positive glioblastoma cells (71).