Hepatic steatosis is promoted by hepatic lipid dysregulation, oxidative stress, mitochondrial dysfunction and chronic systemic inflammation otherwise induced by pro-inflammatory cytokines, such as monocyte chemoattractant protein-1 (MCP-1), tumor necrosis factor-α (TNF-α), and interleukin-6 (IL-6), secreted by the liver and adipocytes [7, 8, 9, 10, 11]. This evidence concerns the gene CCL2 and Hepatic steatosis.