Altogether, our results demonstrate that insufficient dietary iron supply can establish molecular features that resemble several neurological disorders, such as abnormal ferritin accumulation in striatum observed in parkinsonian syndromes and Huntington disease (Vidal et al., 2004; Simmons et al., 2007), increased MAO-A activity in prefrontal cortex which is associated with depression (Meyer et al., 2009) and increased degree of lipid peroxidation in hippocampus that is related with most of conformational diseases (Sultana et al., 2013). This evidence concerns the gene MAOA and major depressive disorder.