Additionally, these authors demonstrated that binding of the Pb14-3-3 mutant to laminin and fibrinogen was reduced compared to that of the control, which correlated with a significant reduction of the virulence phenotype in the invertebrate infection model Galleria mellonella. This study established multifaceted roles of Pb14-3-3 in morphology, attachment/infection to host components, and virulence, therefore supporting the previous report that suggested 14-3-3 as interesting therapeutic target for the treatment of PCM. Here, LAMB2 is linked to infection.