NR1H3 and steatosis: We demonstrate that steatosis may be induced in B-13/H cells through exposure to high concentrations of fatty acids; that B-13/H cells express transcriptionally functional LXRα; that B-13/H cells are susceptible to steatosis after treatment with LXR activators and that exposing B-13/H cells to drugs which cause phospholiposis results in a dose- and time-dependent increase in phospholipid accumulation.