CSF3 and myocardial infarction: We could demonstrate that a dual therapy consisting of (stem) cell mobilization with granulocyte-colony stimulating factor (G-CSF) and stabilization of SDF-1 by preventing its cleavage through inhibition of the protease CD26 increased recruitment of blood-derived progenitor cells associated with attenuated post-MI remodeling, improved myocardial function, and increased survival in mice [4].