Of note, the ratio of M2-like CXCR4-EGFP+CD11b+/CD206+ cells to inflammatory M1-like CD11b+/CD86+ cells increased by 2.6-fold after DMOG-induced PH inhibition compared to infarcted control mice, suggesting an improved fine-tuning of reparative M2-like cells in the ischemic heart after 7 days. Here, CD86 is linked to benign neoplasm.