NEAT1 was also reported to be a well sponge platform for many kinds of miRNA, such as miR-548 in the regulation of breast cancer cell apoptosis [51], miR-204 in regulation of epithelial-to-mesenchymal transition (EMT) and the radioresistance of NPC cells [52], and has-mir-98-5p in regulation of EGCG-induced CTR1 and cDDP sensitivity enhancement in NSCLC [53]. This evidence concerns the gene NEAT1 and breast carcinoma.