The selective ion channel inhibitions considered in this study yielded similar propagation patterns qualitatively consistent between virtual AP phenotypes: inhibition of IK1, INaK, or INa entailed a more notable wave meandering that reduces the organization of the arrhythmia for all the AP phenotypes, either by slowing down electrical propagation in the human atria (INa block) or enlarging the propagation wavefront (IK1 and INaK block). This evidence concerns the gene INA and cardiac arrhythmia.