Alveolar epithelial cells may promote to inflammatory events in ALI/acute respiratory distress sydrome (ARDS) and are an important source of cytokines (eg. TNF-a, IL-1b, IL-6) and chemokines (eg. monocyte chemotactic protein MCP-1, IL8) under inflammatory Conditions [27–29]. This evidence concerns the gene IL6 and acute respiratory distress syndrome.