We also identified novel associations with genes that code for a transcription factor (FOXP4) that validated in clinical ILD cases, a cell adhesion molecule (ALCAM) expressed in the pulmonary microvascular endothelium, a protein involved in reorganization of the actin cytoskeleton (DAAM1) that may play a role in pulmonary vascular remodeling, and a (STK38) MAP kinase inhibiting protein that protects against acute lung injury. The gene discussed is DAAM1; the disease is interstitial lung disease.