This discovery came as a result of follow-up on our GWAS finding that HAA, a putative measure of subclinical ILD, is associated with genetic variation at rs7852363, a SNP that sits within the long non-coding RNA FLJ35282 downstream of the CDKN2A/CDKN2B/ANRIL locus. The gene discussed is CDKN2B-AS1; the disease is interstitial lung disease.