More specifically, NF-κB signaling was recently assessed in cervical cancer and epidermoid carcinoma cells to inhibit apoptosis and mediate CP resistance.31, 32 However, the mediator between chemotherapy agent and NF-κB activity remains poorly understood, making it difficult to identify the target that could re-sensitize those resistant tumor cells to cytotoxic agents. The gene discussed is NFKB1; the disease is neoplasm.