Based on the various literatures, different mechanisms underlying the development of cervical carcinoma were proposed, mainly including phosphatidylinositol-3 kinase (PI3K)/Akt/mTOR [44–46], the mitogen-activated protein kinases (MAPKs) extracellular signal-related kinase (ERK1/2) [47, 48], Janus kinase 2 (JAK2)/signal transducer and activator of transcription-3 (STAT3) [49], JAK3/STAT5 [50], NF-κB [51], Wnt/β-catenin [52], and c-Jun N-terminal kinase (JNK)/p-38 [53] signaling pathways. The gene discussed is STAT3; the disease is cervical carcinoma.