To further confirm that BMX promotes cell proliferation and tumor formation through the AKT/mTOR pathway in cervical cancer cells, the AKT inhibitor MK-2206 and mTOR inhibitor rapamycin were used in HeLa-wt, HeLa-BMX+/−, SiHa-shGFP and SiHa-shBMX cell lines. The gene discussed is AKT1; the disease is neoplasm.