In contrast, in a previous study investigating the differential expression of arginase and inducible NOS (iNOS) in the lungs in a CLP-sepsis rodent model, the authors demonstrated that strong expression of iNOS was found in alveolar, bronchial epithelial cell, endothelial cells, and alveolar macrophages, whereas arginase was almost undetectable after CLP [36]. The gene discussed is NOS2; the disease is Sepsis.