In previous studies, CHD showed anti-inflammatory effects by reducing NO induced by cyclooxygenase-2 (COX-2), interferon gamma (IFN-r), interleukin 4 (IL-4), tumor necrosis factor alpha (TNF-a), prostaglandin E2 (PGE-2), vascular cell adhesion molecule 1 (VCAM-1), monocyte chemoattractant protein-1 (MCP-1), and inducible NOS (iNOS) [4–6]. The gene discussed is IL4; the disease is coronary artery disorder.