Furthermore, our study (Kalu W. et al., 2016) on animal models of BPH showed significant elevation of prostatic lipid peroxidation with concomitant significant reduction of the prostatic levels of GSH, SOD, GPx, and catalase activities of BPH untreated rats and which parameters were significantly improved following treatment with finasteride or kolaviron. This evidence concerns the gene CAT and benign prostatic hyperplasia.