Glut1 and HK-II have been identified in our previous work, and HF downregulated the expressions of both targets.15 Here, we found that both Glut1 and HK-II were enhanced in Atg7−/− MEFs to support our notion that Atg7 is required for aerobic glycolysis modulation during nutrient-replete condition (Figure 5b). The gene discussed is ATG7; the disease is hydrops fetalis.