Consistent with this, mouse models of ALS with SOD1, TDP43, FUS, or C9ORF72 mutations also display NMJ defects (Dadon‐Nachum et al, 2011; Arnold et al, 2013; Liu et al, 2016; Sharma et al, 2016). Here, C9orf72 is linked to amyotrophic lateral sclerosis.