Our microarray data revealed a major suppression of several components of the JNK pathway (MAP4K2, MAP3K5, MAP2K7 and MAP2K4), with the exception of JNK2 (also known as MAPK9), which was increased at 24 h (Supplementary Data 3, Fig. 5); the other two Jun kinases, JNK1 and JNK3, were not significantly affected by infection. The gene discussed is MAPK10; the disease is infection.