Multiple acyl-CoA dehydrogenase deficiency (MADD), also called as glutaric aciduria type II, ethylmalonic-adipic aciduria, and riboflavin-responsive C6-C10 dicarboxylic aciduria, is caused due to the deficiency of the electron transfer flavoprotein (ETF) or its dehydrogenase and ubiquinone oxidoreductase (ETF-QO) [23]. The gene discussed is TEAD2; the disease is multiple acyl-CoA dehydrogenase deficiency.