On the other hands, it has been investigated that overexpression of vascular endothelial growth factor (VEGF) which was shown in about 20% of HCC patients tended to result in shortened overall survival [17], and inhibition and MET in MET-positive HCC which consist about 30-40% of HCC patients resulted in decreased tumor burden [28, 29]. The gene discussed is MET; the disease is neoplasm.