OCLN and infection: Increased susceptibility to infection and a striking in vivo change in occludin localization to the apical TJ was identified in mice lacking a specific γδT-cellpopulation of iIELs whereas in γδiIEL knockout mice no such redistribution occurred implying that γδiIEL interaction with SI epithelial cells played a central role in maintaining TJ integrity and barrier function in response to T. gondii infection.60