Mice with the NMDA subunit 1 receptor selectively removed from parvalbumin-expressing GABAergic interneurons show a similar EEG profile to Fmr1 KO mice, including increased basal gamma EEG, and display several autism-like behavioral changes (Carlén et al. 2012; Saunders et al. 2013; Billingslea et al. 2014). This evidence concerns the gene FMR1 and autism.