The levels of these receptors especially that of SLC1A5 are found to be overexpressed in breast and prostate cancer cell lines and pharmacological inhibitors such as benzylserine (BenSer) and l-γ-glutamyl-p-nitroanilide (GPNA) or shRNA-mediated inactivation/suppression of the glutamine transporter has been found to stall the proliferation of tumor cells (73, 74) (Table 1). This evidence concerns the gene SLC1A5 and neoplasm.