However, despite a clear induction of IL-17A expression promoted by a peritoneal inoculation of A. baumannii, the neutralization of this cytokine by antibodies during septicemia, or infection in a IL-17A-deficient background, neither affected bacterial burden nor survival rate, when compared with antibody control-treated mice or wild-type (WT) mice (18). The gene discussed is IL17A; the disease is Sepsis.