In coordination with MuSK, the fetal nAChR γ-subunit [72] (antibodies to this subunit are detected in fetal nAChR inactivation syndrome, FARIS [73]), and the L-type Ca2+ channel dihydropyridine receptor [74] (antibodies to this receptor are detected in some MG patients [75]) are important in muscle prepatterning of AChR clusters which participate in axonal guidance to the muscle [1,2,14,17,29]. This evidence concerns the gene MUSK and myasthenia gravis.