One animal study showed that T. cruzi-infected CBA mice treated with cromolyn (a MC stabilizer) presented much greater parasitemia and IFN-γ levels, higher mortality, myocarditis, and cardiac damage, indicating that MCs control blood and tissue parasitemia, IFN-γ production, cardiac inflammation, and susceptibility to infection, suggesting that MCs are involved in resistance to this infection (36). The gene discussed is IFNG; the disease is myocarditis.